carotid artery primary endothelial cells ecs Search Results


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ATCC human coronary artery endothelial cells hcaecs
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Cell Applications Inc human carotid artery endothelial cells hctaecs
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PromoCell hcaecs
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ATCC bovine pulmonary arterial endothelial cells
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Vec Technologies primary bovine pulmonary artery endothelial cells
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ScienCell human coronary artery endothelial cells (hcaecs)
Human Coronary Artery Endothelial Cells (Hcaecs), supplied by ScienCell, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Cell Applications Inc human coronary artery endothelial cells
Human Coronary Artery Endothelial Cells, supplied by Cell Applications Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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PromoCell primary endothelial cells
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Cambrex human umbilical arterial endothelial cells (huaecs)
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Thermo Fisher bpaec cells
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Cell Systems Corporation human cerebral artery microvascular endothelial cells
Carvacrol stimulates TRPV3 cation currents in cerebral artery <t>endothelial</t> cells A, representative whole-cell currents recorded from human <t>microvascular</t> cerebral artery endothelial cells during voltage ramps from −100 to +100 mV. Current density is shown for cells under baseline conditions (B) and after first (1), second (2), and third (3) application of carvacrol (100 μM). B, summary data demonstrating sensitization of the carvacrol-induced current (n = 6). C, summary data showing the effects of the TRPA1 antagonist HC-030031 (HC, 3 μM) and the TRPV1–4 blocker RuR (10 μM) on carvacrol-induced currents recorded from cerebral artery endothelial cells; n = 5 for HC-030031, n = 4 for ruthenium red. Current magnitude was normalized to peak carvacrol-induced currents. *, P ≤ 0.05 versus control (C).
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Lonza human pulmonary artery endothelial cells
Carvacrol stimulates TRPV3 cation currents in cerebral artery <t>endothelial</t> cells A, representative whole-cell currents recorded from human <t>microvascular</t> cerebral artery endothelial cells during voltage ramps from −100 to +100 mV. Current density is shown for cells under baseline conditions (B) and after first (1), second (2), and third (3) application of carvacrol (100 μM). B, summary data demonstrating sensitization of the carvacrol-induced current (n = 6). C, summary data showing the effects of the TRPA1 antagonist HC-030031 (HC, 3 μM) and the TRPV1–4 blocker RuR (10 μM) on carvacrol-induced currents recorded from cerebral artery endothelial cells; n = 5 for HC-030031, n = 4 for ruthenium red. Current magnitude was normalized to peak carvacrol-induced currents. *, P ≤ 0.05 versus control (C).
Human Pulmonary Artery Endothelial Cells, supplied by Lonza, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Carvacrol stimulates TRPV3 cation currents in cerebral artery endothelial cells A, representative whole-cell currents recorded from human microvascular cerebral artery endothelial cells during voltage ramps from −100 to +100 mV. Current density is shown for cells under baseline conditions (B) and after first (1), second (2), and third (3) application of carvacrol (100 μM). B, summary data demonstrating sensitization of the carvacrol-induced current (n = 6). C, summary data showing the effects of the TRPA1 antagonist HC-030031 (HC, 3 μM) and the TRPV1–4 blocker RuR (10 μM) on carvacrol-induced currents recorded from cerebral artery endothelial cells; n = 5 for HC-030031, n = 4 for ruthenium red. Current magnitude was normalized to peak carvacrol-induced currents. *, P ≤ 0.05 versus control (C).

Journal: Molecular Pharmacology

Article Title: A Dietary Agonist of Transient Receptor Potential Cation Channel V3 Elicits Endothelium-Dependent Vasodilation S⃞

doi: 10.1124/mol.109.060715

Figure Lengend Snippet: Carvacrol stimulates TRPV3 cation currents in cerebral artery endothelial cells A, representative whole-cell currents recorded from human microvascular cerebral artery endothelial cells during voltage ramps from −100 to +100 mV. Current density is shown for cells under baseline conditions (B) and after first (1), second (2), and third (3) application of carvacrol (100 μM). B, summary data demonstrating sensitization of the carvacrol-induced current (n = 6). C, summary data showing the effects of the TRPA1 antagonist HC-030031 (HC, 3 μM) and the TRPV1–4 blocker RuR (10 μM) on carvacrol-induced currents recorded from cerebral artery endothelial cells; n = 5 for HC-030031, n = 4 for ruthenium red. Current magnitude was normalized to peak carvacrol-induced currents. *, P ≤ 0.05 versus control (C).

Article Snippet: Carvacrol-activated currents were recorded from human cerebral artery microvascular endothelial cells (Cell Systems Corporation, Kirkland, WA).

Techniques: Control